By Albert Abrams
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Extra info for Spondylotherapy; physio-therapy of the spine based on a study of clinical physiology
After hyperoxic exposure (or normoxia), all mothers and pups were returned to (or maintained at) room air, and returned to standard animal quarters until studied at 3 to 5 months of age. 0 g (mean ± SEM, n = 18) for control vs. 0 g for PNH-treated rats (n = 32) (NS). Methods and limitations of CSN recording under general anesthesia have been described in detail elsewhere (Bisgard et al. 2003). 3 Results The responses to NaCN injection and to sustained 100% N2 (max N2) in a normal control rat are presented in Fig.
A. (1991) The coupling of ventilation to pulmonary gas exchange during exercise. J. Whipp and K. ), Pulmonary Physiology and Pathophysiology of Exercise. Dekker, New York, pp. 271–307. Part II Oxygen Sensing and the Carotid Body 4 A Peripheral Oxygen Sensor Provides Direct Activation of an Identified Respiratory CPG Neuron in Lymnaea Harold J. Bell1, Takuya Inoue2 and Naweed I. Syed3 Abstract The mechanisms by which peripheral, hypoxia-sensitive chemosensory cells modulate the output from the respiratory central pattern generator (CPG) remain largely unknown.
Vco2 kinetics are speeded both in absolute terms and relative to those of Vo2, . presumably because the . muscle CO2 capacitance is already partially charged. V e “tracks” the altered V co2 . . kinetics such that the linear steady-state Ve-Vco2 relationship (with a positive Ve-intercept) is main. tained throughout each WR transient despite the markedly-changed V co2 kinetics. This demonstrates that if Ve is, somehow “CO2 coupled,” it is not with the muscle CO2 production rate but with its exchange rate at the lung.
Spondylotherapy; physio-therapy of the spine based on a study of clinical physiology by Albert Abrams